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Hepatitis B

Potent antiviral therapies and liver-supportive agents to manage acute and chronic Hepatitis B infections safely and effectively.

  • Hepatitis B is a viral infection of the liver caused by the Hepatitis B virus (HBV). It can present as a short-term (acute) illness or lifelong (chronic) infection. Chronic HBV increases risk of liver inflammation, fibrosis, cirrhosis, and hepatocellular carcinoma if not adequately managed.

    • Acute Phase (may be asymptomatic):

      • Fatigue, low-grade fever, joint pain

      • Jaundice (yellowing of skin/eyes)

      • Dark urine, pale stools

      • Abdominal discomfort (right upper quadrant)

      • Chronic Phase (often silent):

    • Mild liver enzyme elevations on blood tests

      • Occasional fatigue or malaise

      • Signs of advanced disease: ascites, spider angiomas, easy bruising

    • Bloodborne Transmission: Shared needles, transfusions with unscreened blood

    • Sexual Contact: Unprotected intercourse with an infected partner

    • Perinatal Transmission: From mother to newborn during delivery

    • Occupational Exposure: Healthcare workers via needlestick injuries

    • Household Contact: Sharing razors or toothbrushes in rare cases

    • Immunosuppression: Chemotherapy, corticosteroids, biologic therapies

    • Co-Infections: HIV or Hepatitis C can accelerate HBV replication

    • High Alcohol Intake: Exacerbates liver damage and viral activity

    • Poor Glycemic Control: Diabetes may impair immune response to HBV

    1. Serologic Markers:

      • HBsAg (Hepatitis B surface antigen): Indicates active infection

      • Anti-HBc IgM: Marker of acute or recent infection

      • Anti-HBs: Confirms immunity (past infection or vaccination)

    2. Viral Load Testing: HBV DNA quantification to assess replication

    3. Liver Function Tests (LFTs): ALT/AST levels gauge inflammation

    4. Ultrasound & Elastography: Evaluate liver fibrosis and cirrhosis

    5. Liver Biopsy (select cases): Histology for staging and treatment decisions

    • Nucleos(t)ide Analogues (First-Line):

      • Tenofovir disoproxil fumarate (300 mg once daily)

      • Entecavir (0.5 mg once daily)
        ― Both offer high barrier to resistance and potent viral suppression.

    • Other Oral Agents:

      • Lamivudine, Telbivudine: Lower barrier—reserved when cost is a major constraint.

    • Immune Modulator:

      • Pegylated Interferon-α2a (180 μg weekly) for a finite course in select patients; can achieve seroconversion.

    • Supportive Liver Care:

      • Ursodeoxycholic Acid, Antioxidants (e.g., silymarin) to support hepatocyte health.

    • Monitoring Protocol:

      • Regular HBV DNA and LFTs every 3–6 months

      • Surveillance ultrasound every 6 months in cirrhosis

  • Q1: How long will I need antiviral therapy?
    A: Most chronic HBV patients require long-term (often indefinite) treatment to maintain viral suppression and prevent relapse.

    Q2: Can Hepatitis B be cured?
    A: A true “cure” (HBsAg loss) is rare; treatment aims for sustained viral suppression and “functional cure.” Research into novel therapies is ongoing.

    Q3: Are there side effects to these antivirals?
    A: Tenofovir and entecavir are generally well-tolerated; potential effects include mild GI upset or headache. Renal function and bone density should be monitored with tenofovir.

     

    Q4: Can I stop medication once my viral load is undetectable?
    A: Stopping therapy risks viral rebound; any discontinuation must be under specialist guidance with close follow-up.

    Q5: How can I prevent spreading HBV?
    A: Practice safe sex, avoid sharing needles or personal items, ensure household members are vaccinated, and follow infection-control precautions.

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